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Additional info for Annual Review of Immunology Volume 14 1996

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61. 62. 63. 64. 65. 66. 67. 68. 69. 70. 71. mational change in T-cell receptors: role in activation and in repertoire selection. Cold Spring Harbor Symp. Quant. Biol. 54:657–66 Matsui K, Boniface JJ, Steffner P, Reay PA, Davis MM. 1994. Kinetics of Tcell receptor binding to peptide/I-Ek complexes: correlation of the dissociation rate with T-cell responsiveness. Proc. Natl. Acad. Sci. USA 91:12862–66 Valitutti S, M¨uller S, Cella M, Padovan E, Lanzavecchia A. 1995. Serial triggering of many T-cell receptors by a few peptideMHC complexes.

35. shall M, Herbert E, McKenzie D, Grey HM, Sette A. 1993. Effect of T-cell receptor antagonism on interaction between T cells and antigen-presenting cells and on T-cell signaling events. Proc. Natl. Acad. Sci. USA 90:2671–75 Sloan-Lancaster J, Shaw AS, Rothbard JB, Allen PM. 1994. Partial T cell signaling: altered phospho-ζ and lack of Zap 70 recruitment in APL-induced T cell anergy. Cell 79:913–22 Bertoletti A, Sette A, Chisari FV, Penna A, Levrero M, De Carli M, Fiaccadori F, Ferrari C. 1994.

Chem. 267:13656–60 40. Valius M, Bazenet C, Kazlauskas A. 1993. Tyrosines 1021 and 1009 are phosphorylation sites in the carboxy terminus of the platelet-derived growth factor receptor beta subunit and are required for binding of phospholipase C gamma and a 64kilodalton protein respectively. Mol. Cell. Biol. 13:133–43 41. Kazlauskas A, Kashishian A, Cooper JA, Valius M. 1992. GTPase-activating protein and phosphatidylinositol 3-kinase bind to distinct regions of the plateletderived growth-factor receptor betasubunit.

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